1. CORTÉS VA, CURTIS DE, SUKUMARAN S,. SHAO X, RASHID S, SMITH AR, REN J,
HAMME RE, AGARWAL AK, HORTON JD, GARG A. MOLECULAR MECHANISMS OF HEPATIC STEATOSIS AND INSULIN RESISTANCE IN THE AGPAT2-DEFICIENT MOUSE MODEL OF CONGENITAL GENERALIZED LIPODYSTROPHY. (CELL METABOLISM, 9:165- 176, 2009).
2. RASHID S, MARCIL M, RUEL I, AND GENEST J. IDENTIFICATION OF A NOVEL LIPID EFFLUX DEFECT CAUSING LOW HDL CHOLESTEROL THAT IS NOT DUE TO MUTATIONS IN THE ABCA1 GENE BUT TO REGULATION OF ABCA1 PROTEIN. (SUBMITTED TO EUROPEAN HEART JOURNAL).
3. RASHID S, ANDERSON NN, HO YK, HAMMER RE, MOON YA AND HORTON JD. THE IN VIVO EFFECTS OF CONDITIONAL GENE DELETION OF SREBP-2. (IN PREPARATION).
4. RASHID SAND FRANCIS G. STATINS AND PRIMARY PREVENTION: IS ALL THE EVIDENCE IN? CANADIAN JOURNAL OF CARDIOLOGY, 24(4): 301-303, 2008.
5. RASHID S AND GENEST J. THE EFFECT OF OBESITY ON HDL METABOLISM. OBESITY RESEARCH, 15(12): 2875-2888, 2007.
6. RASHID S AND KHANDAKER NR. RISK FACTORS FOR EARLY MYOCARDIAL INFARCTION IN SOUTH ASIANS.JAMA, 297(17): 1880-1881, 2007.
7. RASHID S. SHOULD CHOLESTEROL-LOWERING MEDICATION BE AVAILABLE WITHOUT A PRESCRIPTION IN CANADA? CANADIAN JOURNAL OF CARDIOLOGY, 23(3): 189- 193, 2007.
8. RASHID S, PATTERSON B AND LEWIS G. WHAT HAVE WE LEARNED ABOUT HDL METABOLISM FROM KINETIC STUDIES IN HUMANS? JOURNAL OF LIPID RESEARCH, 47(8): 1631-42, 2006.
9. KRIMBOU L, HASSAN HH, BLAIN S, RASHID S, DENIS M, MARCIL M AND GENEST J. BIOGENESIS AND SPECIATION OF NASCENT APOA-I-CONTAINING PARTICLES IN VARIOUS CELL LINES. JOURNAL OF LIPID RESEARCH, 46: 1668-1677, 2005.
10. RASHID S, CURTIS DE, GARUTI R, ANDERSON NN, BASHMAKOV Y, HO YK, HAMMER
RE, MOON YA AND HORTON JD. DECREASED PLASMA CHOLESTEROL AND HYPERSENSITIVITY TO STATINS IN MICE LACKING PCSK9. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES, 102(15), 2005.
11. RASHID S AND LEWIS, GF. THE MECHANISMS OF DIFFERENTIAL MINERALOCORTICOID AND GLUCOCORTICOID ACTION IN THE BRAIN AND PERIPHERAL TISSUES. CLINICAL BIOCHEMISTRY, 38: 401-09, 2005.
12. RASHID S, WATANABE T, SAKAUE T AND LEWIS GF. MECHANISMS OF HDL LOWERING IN INSULIN-RESISTANT, HYPERTRIGLYCERIDEMIC STATES: THE COMBINED EFFECT OF HDL TRIGLYCERIDE-ENRICHMENT AND ELEVATED HEPATIC LIPASE ACTIVITY. CLINICALBIOCHEMISTRY , 36(6): 421-29, 2003.
13. RASHID S, TRINH DK, UFFELMAN KD, COHN, JS, RADER DJ AND LEWIS GF. EXPRESSION OF HUMAN HEPATIC LIPASE IN THE RABBIT MODEL PREFERENTIALLY ENHANCES THE CLEARANCE OF TRIGLYCERIDE ENRICHED VERSUS NATIVE HDL APOLIPOPROTEIN A-I. CIRCULATION, 107(24): 3066-72, 2003.
14. RASHID S, BARRETT PHR, UFFELMAN KD, WATANABE T, ADELI K, AND LEWIS GF. LIPOLYSIS OF TRIGLYCERIDE-ENRICHED HDL BY HEPATIC LIPASE EX-VIVO ENHANCES HDL APOLIPOPROTEIN A-I CLEARANCE IN A RABBIT MODEL. ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY, 22(3):483-7, 2002.
15. RASHID S, UFFELMAN KD, BARRETT PHR, AND LEWIS GF. THE EFFECT OF ATORVASTATIN ON HDL APOLIPOPROTEIN A-I PRODUCTION AND CLEARANCE IN THE NEW ZEALAND WHITE RABBIT.CIRCULATION, 106(23): 2955-60, 2002.
16. RASHID S, UFFELMAN KD, AND LEWIS GF. THE MECHANISM OF HDL LOWERING IN HYPERTRIGLYCERIDEMIC, INSULIN RESISTANT STATES. JOURNAL OF DIABETES AND ITS COMPLICATIONS, 16(1):24-8, 2002.
17. RASHID S, UFFELMAN KD, BARRETT PHR, VICINI P, ADELI K, AND LEWIS
GF.TRIGLYCERIDE ENRICHMENT OF HDL DOES NOT ALTER HDL-SELECTIVE CHOLESTERYL ESTER CLEARANCE IN RABBITS. JOURNAL OF LIPID RESEARCH , 42(2):265-71, 2001.
18. LEWIS GF, RASHID S, AND LAMARCHE B. MECHANISM OF HDL LOWERING IN INSULIN RESISTANT STATES. ADV EXP MED BIOL, 498:273-7, 2001.
19. RASHID S, SHI Z.Q, NIWA M, MATHOO JMR, VAN DELANGERYT M, BILINSKI D, LEWIS
GF, AND VRANIC M. BETA-BLOCKADE, BUT NOT NORMOGLYCEMIA NOR HYPERINSULINEMIA, MARKEDLY DIMINISHES STRESS-INDUCED HYPERGLYCEMIA IN DIABETIC DOGS. DIABETES , 49(2):253-62, 2000.
20. LAMARCHE, B, RASHID S, AND LEWIS G.F. HDL METABOLISM IN HYPERTRIGLYCERIDEMIC STATES: AN OVERVIEW. CLIINICA CHIMICA ACTA. 286(1- 2):145-61, 1999.
21. SADHU H, WIESENTHAL SR, MACDONALD PE, MCCALL RH, TCHIPASHVILI V,
RASHID S, SATKUNARAJAH M, IRWIN DM, SHI ZQ, BRUBAKER, PL., WHEELER MB, VRANIC M, EFENDIC S. AND GIACCA A. GLUCAGON-LIKE PEPTIDE 1 INCREASES INSULIN SENSITIVITY IN DEPANCREATIZED DOGS. DIABETES 48(5):1045-53, 1999.
22. NIWA M, RASHID S, SHUM K, MATHOO J, TCHIPAHVILI V, MATHOO J, CHAN O,
KAWAMORI R, VRANIC M, AND GIACCA A. THE EFFECT OF JTT501 ON HEPATIC AND PERIPHERAL GLUCOSE UPTAKE IN ALLOXAN-INDUCED DIABETIC DOGS. METABOLISM 49(7): 862-7, 2000. PUBLICATIONS IN SUPPLEMENTS
23. RASHID S, RUEL I, MARCIL M AND GENEST J. IDENTIFICATION OF A NOVEL LIPID EFFLUX DEFECT CAUSING LOW HDL CHOLESTEROL THAT IS NOT DUE TO MUTATIONS IN THE ABCA1 GENE BUT TO REGULATION OF ABCA1 PROTEIN. CIRCULATION 116 (SUPPL.II) (16): 1179, 2007.
24. RASHID S, RUEL I, ALRASSADI K, DASTANI Z, MARCIL M AND GENEST J. CELLULAR PHYSIOLOGY OF CHOLESTEROL EFFLUX DEFECTS IN LOW HDL-C INDIVIDUALS WITH A NORMAL ABCA1 GENE. CIRCULATION 114 (SUPPL. II) (18): 1347, 2006.
25. DENIS M, HASSAN HH, RASHID S, KRIMBOU AND GENEST J. FORMATION OF A PRODUCTION COMPLEX BETWEEN APOLIPOPROTEIN A-I AND THE ATP-BINDING CASSETTE TRANSPORTER A1 IS ESSENTIAL FOR INTRACELLULAR LIPIDATION OF APOA-I. CIRCULATION 112 (SUPPL. S) (17): U158 584, 2005.
26. RASHID S, TRINH D, UFFELMAN K, RADER D, LEWIS GF. EXPRESSION OF HUMAN HEPATIC LIPASE IN THE RABBIT MODEL PREFERENTIALLY ENHANCES THE CLEARANCE OF TRIGLYCERIDE-ENRICHED VERSUS NATIVE HDL APOLIPOPROTEIN A- I. DIABETES 52 (SUPPL. 1): A73, 2003.
27. NIWA M, RASHID S, SHUM K, MATHOO J, TCHIPACHVILI V, CHAN O, KAWAMORI R,
VRANIC M, GIACCA A. JTT-501 INCREASES HEPATIC AND PERIPHERAL GLUCOSE UPTAKE AND SUPPRESSES GLUCOSE PRODUCTION IN ALLOXAN-INDUCED DIABETIC DOGS. DIABETES 47 (SUPPL. 1):A94, 1998.
28. SANDHU H, WIESENTHAL S, MCCALL R, TCHIPACHVILI V, RASHID S, VRANIC M,
EFENDIC S, GIACCA A. GLUCAGON-LIKE PEPTIDE 1 (GLP-1) INCREASES INSULIN SENSITIVITY IN DIABETIC DOGS. DIABETES 46 (SUPPL. 1): 29A, 1997.
29. RASHID S, NIWA M, VANDELANGERYT M, MATHOO J, BILINSKI D, SHI Q, VRANIC M. THE IMPAIRMENT OF THE GLUCOREGULATORY RESPONSES TO STRESS IN DIABETES. DIABETES 46 (SUPPL. 1); 230A, 1997.
NON-PEER-REVIEWED PUBLICATIONS
30. RASHID S AND LEWIS GF. THE CONTRIBUTIONS OF TRIGLYCERIDE ENRICHMENT OF HDL AND LIPOLYSIS OF HDL BY HEPATIC LIPASE IN THE PATHOPHYSIOLOGICAL LOWERING OF HDL CONCENTRATIONS IN HYPERTRIGLYCERIDEMIC STATES. INTERNATIONAL ATHEROSCLEROSIS WEBSITE, 2002.
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7. The only amino acid that does not need to enter the A site before entering the P site on a ribosome during the process of translation is methionine. Methionine is coded for by AUG, the start codon, and therefore it is always the first amino acid in a newly synthesized polypeptide. Since it is the first amino acid, there will be no amino acid before it to form a peptide bond with and hence it n